About: Autophagy and Viral Infection

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An Entity of Type : schema:ScholarlyArticle, within Data Space : covidontheweb.inria.fr associated with source document(s)

Attributes	Values
type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
has title	Autophagy and Viral Infection
Creator	Tan, Peng He, Lian Zhou, Yubin Lin, Eena Mao, Jingrong Yu, Jiaming
Source	Medline; PMC
abstract	Autophagy is an intracellular recycling process that maintains cellular homeostasis by orchestrating immunity upon viral infection. Following viral infection, autophagy is often initiated to curtail infection by delivering viral particles for lysosomal degradation and further integrating with innate pattern recognition receptor signaling to induce interferon (IFN)-mediated viral clearance. However, some viruses have evolved anti-autophagy strategies to escape host immunity and to promote viral replication. In this chapter, we illustrate how autophagy prevents viral infection to generate an optimal anti-viral milieu, and then concentrate on how viruses subvert and hijack the autophagic process to evade immunosurveillance, thereby facilitating viral replication and pathogenesis. Understanding the interplays between autophagy and viral infection is anticipated to guide the development of effective anti-viral therapeutics to fight against infectious diseases.
has issue date	2019-11-15(xsd:dateTime)
bibo:doi	10.1007/978-981-15-0606-2_5
bibo:pmid	31728865
has license	no-cc
sha1sum (hex)	f75eea18b2baa316b9aab11db18654aba56d0577
schema:url	https://doi.org/10.1007/978-981-15-0606-2_5
resource representing a document's title	Autophagy and Viral Infection
has PubMed Central identifier	PMC7122562
has PubMed identifier	31728865
schema:publication	Autophagy Regulation of Innate Immunity
resource representing a document's body	covid:f75eea18b2baa316b9aab11db18654aba56d0577#body_text
is schema:about of	named entity 'escape' named entity 'autophagy' named entity 'viral infection' named entity 'host' named entity 'Autophagy' named entity 'Viral' named entity 'Infection' named entity 'GENERATE' named entity 'INTEGRATING' named entity 'STRATEGIES' named entity 'PATHOGENESIS' named entity 'EVOLVED' named entity 'VIRAL REPLICATION' named entity 'CELLULAR HOMEOSTASIS' named entity 'VIRAL PARTICLES' named entity 'CHAPTER' named entity 'AUTOPHAGY' named entity 'RECEPTOR SIGNALING' named entity 'AUTOPHAGY' named entity 'VIRAL INFECTION' named entity 'VIRUSES' named entity 'IMMUNOSURVEILLANCE' named entity 'VIRAL CLEARANCE' named entity 'ANTICIPATED' named entity 'TO GUIDE' named entity 'PREVENTS' named entity 'ESCAPE' named entity 'DEVELOPMENT' named entity 'HAVE' named entity 'FIGHT' named entity 'THERAPEUTICS' named entity 'FACILITATING' named entity 'CONCENTRATE' named entity 'VIRAL INFECTION' named entity 'EFFECTIVE' named entity 'INTRACELLULAR' named entity 'PATTERN RECOGNITION RECEPTOR' named entity 'PROMOTE' named entity 'MEDIATED' named entity 'LYSOSOMAL DEGRADATION' named entity 'INFECTION' named entity 'OPTIMAL' named entity 'VIRAL' named entity 'INDUCE' named entity 'HOW' named entity 'HOST IMMUNITY' named entity 'PROCESS' named entity 'IMMUNITY' named entity 'INFECTIOUS DISEASES' named entity 'UNDERSTANDING' named entity 'INTERFERON ' named entity 'RECYCLING PROCESS' named entity 'FOLLOWING' named entity 'viruses' named entity 'Following' named entity 'recycling' named entity 'autophagy' named entity 'viruses' named entity 'IFN' named entity 'homeostasis' named entity 'anticipated' named entity 'innate' named entity 'maintains' named entity 'effective' named entity 'promote' named entity 'particles' named entity 'autophagic' named entity 'autophagy' named entity 'interferon' named entity 'infectious diseases' named entity 'anti-viral' named entity 'cellular homeostasis' named entity 'viral infection' named entity 'viruses' named entity 'viral replication' named entity 'virion particles'

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