About: Commentary on the Regulation of Viral Proteins in Autophagy Process

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An Entity of Type : schema:ScholarlyArticle, within Data Space : covidontheweb.inria.fr associated with source document(s)

Attributes	Values
type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
has title	Commentary on the Regulation of Viral Proteins in Autophagy Process
Creator	Hsu, Wei-Li Liu, Hung-Jen Cheng, Ching-Yuan Chi, Pei-I
Source	Medline; PMC
abstract	The ability to subvert intracellular antiviral defenses is necessary for virus to survive as its replication occurs only in the host cells. Viruses have to modulate cellular processes and antiviral mechanisms to their own advantage during the entire virus life cycle. Autophagy plays important roles in cell regulation. Its function is not only to catabolize aggregate proteins and damaged organelles for recycling but also to serve as innate immunity to remove intracellular pathogenic elements such as viruses. Nevertheless, some viruses have evolved to negatively regulate autophagy by inhibiting its formation. Even more, some viruses have employed autophagy to benefit their replication. To date, there are more and more growing evidences uncovering the functions of many viral proteins to regulate autophagy through different cellular pathways. In this review, we will discuss the relationship between viruses and autophagy and summarize the current knowledge on the functions of viral proteins contributing to affect autophagy process.
has issue date	2014-03-10(xsd:dateTime)
bibo:doi	10.1155/2014/962915
bibo:pmid	24734254
has license	cc-by
sha1sum (hex)	e172d0ebcbb5e8488f19e2193ac9a9ed96f60efb
schema:url	https://doi.org/10.1155/2014/962915
resource representing a document's title	Commentary on the Regulation of Viral Proteins in Autophagy Process
has PubMed Central identifier	PMC3966343
has PubMed identifier	24734254
schema:publication	Biomed Res Int
resource representing a document's body	covid:e172d0ebcbb5e8488f19e2193ac9a9ed96f60efb#body_text
is schema:about of	named entity 'cells' named entity 'TO REGULATE' named entity 'REVIEW' named entity 'DAMAGED' named entity 'CELL' named entity 'CONTRIBUTING' named entity 'THEIR' named entity 'ITS' named entity 'INNATE IMMUNITY' named entity 'TO REMOVE' named entity 'HAVE' named entity 'REGULATION' named entity 'PROTEINS' named entity 'IMPORTANT' named entity 'FUNCTIONS' named entity 'DIFFERENT' named entity 'INHIBITING' named entity 'SURVIVE' named entity 'ELEMENTS' named entity 'UNCOVERING' named entity 'PATHOGENIC' named entity 'CELLULAR PROCESSES' named entity 'ROLES' named entity 'COMMENTARY' named entity 'GROWING' named entity 'AFFECT' named entity 'ANTIVIRAL' named entity 'REPLICATION' named entity 'MECHANISMS' named entity 'RECYCLING' named entity 'RELATIONSHIP' named entity 'VIRUS' named entity 'CURRENT' named entity 'REGULATION' named entity 'VIRAL PROTEINS' named entity 'AUTOPHAGY PROCESS' named entity 'VIRUSES' named entity 'INTRACELLULAR' named entity 'ORGANELLES' named entity 'AUTOPHAGY' named entity 'ADVANTAGE' named entity 'ENTIRE' named entity 'AUTOPHAGY PROCESS' named entity 'DATE' named entity 'REGULATE' named entity 'LIFE CYCLE' named entity 'EVOLVED' named entity 'MODULATE' named entity 'FUNCTION' named entity 'AGGREGATE' named entity 'DISCUSS' named entity 'KNOWLEDGE' named entity 'PATHWAYS' named entity 'EMPLOYED' named entity 'CELLULAR' named entity 'BUT' named entity 'HOST CELLS' named entity 'TO SERVE AS' named entity 'FORMATION' named entity 'PLAYS' named entity 'NECESSARY' named entity 'BENEFIT' named entity 'VIRAL PROTEINS' named entity 'autophagy' named entity 'virus' named entity 'subvert' named entity 'viruses' named entity 'life cycle' named entity 'intracellular' named entity 'cell' named entity 'inhibiting' named entity 'organelles' named entity 'antiviral' named entity 'viruses' named entity 'organelles' named entity 'autophagy' named entity 'catabolize' named entity 'innate immunity'

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