About: Accumulating evidence for a role of oxidized phospholipids in infectious diseases

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An Entity of Type : schema:ScholarlyArticle, within Data Space : covidontheweb.inria.fr associated with source document(s)

Attributes	Values
type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
has title	Accumulating evidence for a role of oxidized phospholipids in infectious diseases
Creator	Knapp, Sylvia Martins, Rui Matt, Ulrich Sharif, Omar
Source	Medline; PMC
abstract	Oxidized phospholipids (OxPL) were originally discovered as by-products and mediators of chronic inflammation such as in atherosclerosis. Over the last years, an increasing body of evidence led to the notion that OxPL not only contribute to the pathogenesis of chronic inflammatory processes but in addition play an integral role as modulators of inflammation during acute infections. Thereby, host defense mechanisms involve the generation of oxygen radicals that oxidize ubiquitously present phospholipids, which in turn act as danger-associated molecular patterns (DAMPs). These OxPL-derived DAMPs can exhibit both pro- and anti-inflammatory functions that ultimately alter the host response to pathogens. In this review, we summarize the currently available data on the role of OxPL in infectious diseases.
has issue date	2014-11-20(xsd:dateTime)
bibo:doi	10.1007/s00018-014-1780-3
bibo:pmid	25410378
has license	no-cc
sha1sum (hex)	cf3c99d79ba19d158762e28ad13608aef4aa6b70
schema:url	https://doi.org/10.1007/s00018-014-1780-3
resource representing a document's title	Accumulating evidence for a role of oxidized phospholipids in infectious diseases
has PubMed Central identifier	PMC7079780
has PubMed identifier	25410378
schema:publication	Cell Mol Life Sci
resource representing a document's body	covid:cf3c99d79ba19d158762e28ad13608aef4aa6b70#body_text
is schema:about of	named entity 'by-products' named entity 'oxidized' named entity 'phospholipids' named entity 'evidence' named entity 'INFLAMMATION' named entity 'CONTRIBUTE ' named entity 'ASSOCIATED' named entity 'PATTERNS' named entity 'PATHOGENESIS' named entity 'BUT' named entity 'CHRONIC' named entity 'GENERATION' named entity 'MODULATORS' named entity 'THESE' named entity 'OXIDIZED PHOSPHOLIPIDS' named entity 'OF EVIDENCE' named entity 'PRESENT' named entity 'OXIDIZE' named entity 'MEDIATORS' named entity 'OXYGEN RADICALS' named entity 'NOTION' named entity 'ALTER' named entity 'ROLE' named entity 'ACCUMULATING' named entity 'INFECTIOUS DISEASES' named entity 'ROLE' named entity 'EVIDENCE FOR' named entity 'EXHIBIT' named entity 'PROCESSES' named entity 'DANGER' named entity 'PLAY' named entity 'INCREASING' named entity 'PRO-' named entity 'AVAILABLE' named entity 'REVIEW' named entity 'INTEGRAL' named entity 'OXIDIZED PHOSPHOLIPIDS' named entity 'PATHOGENS' named entity 'ADDITION' named entity 'RESPONSE TO' named entity 'TURN' named entity 'DAMPS' named entity 'DATA' named entity 'FUNCTIONS' named entity 'INFECTIOUS DISEASES' named entity 'CURRENTLY' named entity 'LED' named entity 'PHOSPHOLIPIDS' named entity 'ACT' named entity 'YEARS' named entity 'BODY' named entity 'ACUTE INFECTIONS' named entity 'HOST DEFENSE' named entity 'CHRONIC INFLAMMATION' named entity 'DEFENSE MECHANISMS' named entity 'PRODUCTS' named entity 'INFLAMMATORY' named entity 'ATHEROSCLEROSIS' named entity 'HOST RESPONSE' named entity 'MOLECULAR' named entity 'DERIVED' named entity 'ANTI-INFLAMMATORY' named entity 'inflammation' named entity 'atherosclerosis' named entity 'anti-inflammatory' named entity 'ultimately' named entity 'DAMPs' named entity 'phospholipids' named entity 'pathogens' named entity 'chronic inflammatory' named entity 'inflammation' named entity 'danger-associated molecular patterns' named entity 'TLR9' named entity 'neutrophils' named entity 'pro-inflammatory' named entity 'TLR' named entity 'TLR6' named entity 'macrophages'

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