About: Macrophage Polarization in Inflammatory Diseases

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An Entity of Type : schema:ScholarlyArticle, within Data Space : covidontheweb.inria.fr associated with source document(s)

Attributes	Values
type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
has title	Macrophage Polarization in Inflammatory Diseases
Creator	Chai, Yan-Fen Liu, Yan-Cun Yao, Yong-Ming Zou, Xian-Biao
Source	Medline; PMC
abstract	Diversity and plasticity are two hallmarks of macrophages. M1 macrophages (classically activated macrophages) are pro-inflammatory and have a central role in host defense against infection, while M2 macrophages (alternatively activated macrophages) are associated with responses to anti-inflammatory reactions and tissue remodeling, and they represent two terminals of the full spectrum of macrophage activation. Transformation of different phenotypes of macrophages regulates the initiation, development, and cessation of inflammatory diseases. Here we reviewed the characters and functions of macrophage polarization in infection, atherosclerosis, obesity, tumor, asthma, and sepsis, and proposed that targeting macrophage polarization and skewing their phenotype to adapt to the microenvironment might hold great promise for the treatment of inflammatory diseases.
has issue date	2014-05-01(xsd:dateTime)
bibo:doi	10.7150/ijbs.8879
bibo:pmid	24910531
has license	cc-by-nc-nd
sha1sum (hex)	cae1f2fd5785845caa42d6497361eba46a68f11d
schema:url	https://doi.org/10.7150/ijbs.8879
resource representing a document's title	Macrophage Polarization in Inflammatory Diseases
has PubMed Central identifier	PMC4046879
has PubMed identifier	24910531
schema:publication	Int J Biol Sci
resource representing a document's body	covid:cae1f2fd5785845caa42d6497361eba46a68f11d#body_text
is schema:about of	named entity 'CHARACTERS' named entity 'PLASTICITY' named entity 'GREAT' named entity 'INITIATION' named entity 'ASTHMA' named entity 'MACROPHAGES' named entity 'PHENOTYPE' named entity 'PRO-' named entity 'TREATMENT' named entity 'ATHEROSCLEROSIS' named entity 'RESPONSES' named entity 'TISSUE REMODELING' named entity 'DIFFERENT' named entity 'TRANSFORMATION' named entity 'CESSATION' named entity 'asthma' named entity 'M2 macrophages' named entity 'hallmarks' named entity 'Here' named entity 'activated' named entity 'HOLD' named entity 'HAVE' named entity 'MACROPHAGE ACTIVATION' named entity 'MICROENVIRONMENT' named entity 'PROMISE' named entity 'ACTIVATED MACROPHAGES' named entity 'FULL SPECTRUM' named entity 'MACROPHAGE POLARIZATION' named entity 'PHENOTYPES' named entity 'TARGETING' named entity 'ROLE' named entity 'INFLAMMATORY REACTIONS' named entity 'PROPOSED' named entity 'DIVERSITY' named entity 'SEPSIS' named entity 'INFLAMMATORY' named entity 'CENTRAL' named entity 'OBESITY' named entity 'DEVELOPMENT' named entity 'ADAPT' named entity 'INFLAMMATORY DISEASES' named entity 'ASSOCIATED WITH' named entity 'HERE' named entity 'ANTI-INFLAMMATORY' named entity 'FUNCTIONS' named entity 'INFECTION' named entity 'HOST DEFENSE' named entity 'INFLAMMATORY DISEASES' named entity 'REVIEWED' named entity 'REPRESENT' named entity 'THEIR' named entity 'TUMOR' named entity 'Diversity' named entity 'activation' named entity 'obesity' named entity 'full spectrum' named entity 'macrophages' named entity 'tumor' named entity 'atherosclerosis' named entity 'macrophages' named entity 'atherosclerosis' named entity 'macrophages' named entity 'M1 macrophages' named entity 'macrophage activation' named entity 'tissue remodeling' named entity 'insulin resistance' named entity 'cell lines' named entity 'Macrophage polarization' named entity 'public health' named entity 'parasite' named entity 'inflammatory diseases' named entity 'macrophages' named entity 'outer membrane' named entity 'IRF4' named entity 'TAM' named entity 'CC chemokine ligand' named entity 'low-density lipoprotein' named entity 'NF-κB'

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