About: Extracellular Vesicles in Viral Spread and Antiviral Response

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An Entity of Type : schema:ScholarlyArticle, within Data Space : covidontheweb.inria.fr associated with source document(s)

Attributes	Values
type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
has title	Extracellular Vesicles in Viral Spread and Antiviral Response
Creator	Antonio López-Guerrero, José Bello-Morales, Raquel Ripa, Inés
Source	Medline; PMC
abstract	Viral spread by both enveloped and non-enveloped viruses may be mediated by extracellular vesicles (EVs), including microvesicles (MVs) and exosomes. These secreted vesicles have been demonstrated to be an efficient mechanism that viruses can use to enter host cells, enhance spread or evade the host immune response. However, the complex interplay between viruses and EVs gives rise to antagonistic biological tasks—to benefit the viruses, enhancing infection and interfering with the immune system or to benefit the host, by mediating anti-viral responses. Exosomes from cells infected with herpes simplex type 1 (HSV-1) may transport viral and host transcripts, proteins and innate immune components. This virus may also use MVs to expand its tropism and evade the host immune response. This review aims to describe the current knowledge about EVs and their participation in viral infection, with a specific focus on the role of exosomes and MVs in herpesvirus infections, particularly that of HSV-1.
has issue date	2020-06-08(xsd:dateTime)
bibo:doi	10.3390/v12060623
bibo:pmid	32521696
has license	cc-by
sha1sum (hex)	4af06e2f0d2e4b81fd97682a55647e9eef67debd
schema:url	https://doi.org/10.3390/v12060623
resource representing a document's title	Extracellular Vesicles in Viral Spread and Antiviral Response
has PubMed Central identifier	PMC7354624
has PubMed identifier	32521696
schema:publication	Viruses
resource representing a document's body	covid:4af06e2f0d2e4b81fd97682a55647e9eef67debd#body_text
is schema:about of	named entity 'simplex' named entity 'viruses' named entity 'MVs' named entity 'viruses' named entity 'expand' named entity 'host' named entity 'EVs' named entity 'virus' named entity 'demonstrated' named entity 'HSV-1' named entity 'This' named entity 'Viral' named entity 'infections' named entity 'mediating' named entity 'participation' named entity 'enter' named entity 'immune response' named entity 'HSV-1' named entity 'virus' named entity 'innate immune' named entity 'tropism' named entity 'immune response' named entity 'Antiviral' named entity 'Extracellular Vesicles' named entity 'viruses' named entity 'MVs' named entity 'virions' named entity 'virions' named entity 'dendritic cells' named entity 'LMP1' named entity 'HVEM' named entity 'LMP1' named entity 'bronchoalveolar lavage fluid' named entity 'secretion' named entity 'exosomal' named entity 'HAV' named entity 'exosomes' named entity 'intracellular' named entity 'lysis' named entity 'microRNAs' named entity 'heparan sulfate' named entity 'cell death' named entity 'memory T cell' named entity 'MVBs' named entity 'virions' named entity 'viral transmission' named entity 'Choroid plexus' named entity 'host cell' named entity 'host factors' named entity 'CHO' named entity 'human herpesvirus 6' named entity 'HCMV' named entity 'influenza' named entity 'pathogenesis' named entity 'miRNAs' named entity 'human pathogen' named entity 'glycoprotein' named entity 'EBV' named entity 'Nef protein' named entity 'HSV-1' named entity 'HCMV' named entity 'LMP1' named entity 'tumor cells' named entity 'endocytosis' named entity 'exosomes' named entity 'human T-cell lymphotropic virus' named entity 'glycosaminoglycans' named entity 'host cells' named entity 'MVBs' named entity 'viral antigens' named entity 'miRNA' named entity 'vesicles' named entity 'nuclear envelope' named entity 'herpesviruses' named entity 'HIV-infected patients' named entity 'human cytomegalovirus' named entity 'myelin' named entity 'Dengue virus' named entity 'HSV-1'

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