About: Chapter 186 Angiotensin II Peptides

Facets (new session)
Description
Metadata
Settings
- owl:sameAs
- Inference Rule:

About: Chapter 186 Angiotensin II Peptides Goto Sponge NotDistinct Permalink

An Entity of Type : schema:ScholarlyArticle, within Data Space : covidontheweb.inria.fr associated with source document(s)

Attributes	Values
type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
has title	Chapter 186 Angiotensin II Peptides
Creator	Iwao, Hiroshi Izumi, Yasukatsu
Source	Elsevier; PMC
abstract	ABSTRACT Much evidence supports the notion that angiotensin II (Ang II), the central product of the renin–angiotensin system (RAS), may play a central role not only in the etiology of hypertension but also in the pathophysiology of cardiovascular diseases in humans. Ang II, via the Ang II type 1 receptor, directly causes cellular phenotypic changes and cell growth, regulates the gene expression of various bioactive substances, and activates multiple intracellular signaling cascades in cardiac myocytes and fibroblasts, as well as vascular endothelial and smooth muscle cells. Recently, new factors have been discovered, such as angiotensin-converting enzyme 2, angiotensin-(1-7), and its receptor Mas. This section summarizes the current knowledge about the broad RAS in the pathophysiology of cardiac hypertrophy and remodeling, heart failure, vascular thickening, and atherosclerosis.
has issue date	2013-12-31(xsd:dateTime)
bibo:doi	10.1016/b978-0-12-385095-9.00186-x
has license	els-covid
sha1sum (hex)	27fbfb693135818f95a81d461b54d2d3a359ab1e
schema:url	https://doi.org/10.1016/b978-0-12-385095-9.00186-x
resource representing a document's title	Chapter 186 Angiotensin II Peptides
has PubMed Central identifier	PMC7150204
schema:publication	Handbook of Biologically Active Peptides
resource representing a document's body	covid:27fbfb693135818f95a81d461b54d2d3a359ab1e#body_text
is schema:about of	named entity 'hypertension' named entity 'cellular' named entity 'supports' named entity 'RAS' named entity 'intracellular signaling' named entity 'THICKENING' named entity 'KNOWLEDGE' named entity 'PLAY' named entity 'MUCH' named entity 'RECENTLY' named entity 'ANGIOTENSIN' named entity 'CAUSES' named entity 'SMOOTH MUSCLE CELLS' named entity 'CARDIOVASCULAR DISEASES' named entity 'RENIN-ANGIOTENSIN SYSTEM' named entity 'REMODELING' named entity 'ENDOTHELIAL' named entity 'MAS' named entity 'HAVE' named entity 'CURRENT' named entity 'ITS' named entity 'SECTION' named entity 'PEPTIDES' named entity 'ETIOLOGY' named entity 'RAS' named entity 'SIGNALING CASCADES' named entity 'MULTIPLE' named entity 'BUT' named entity 'ANGIOTENSIN-CONVERTING ENZYME 2' named entity 'VARIOUS' named entity 'SUBSTANCES' named entity 'ACTIVATES' named entity 'NOTION' named entity 'PATHOPHYSIOLOGY' named entity 'EVIDENCE' named entity 'CHANGES' named entity 'DIRECTLY' named entity 'ATHEROSCLEROSIS' named entity 'PRODUCT' named entity 'NEW' named entity 'ABOUT' named entity 'HUMANS' named entity 'BROAD' named entity 'ROLE' named entity 'VASCULAR' named entity 'CARDIAC HYPERTROPHY' named entity 'TYPE 1' named entity 'ANGIOTENSIN II' named entity 'INTRACELLULAR SIGNALING' named entity 'II TYPE' named entity 'GENE EXPRESSION' named entity 'HEART FAILURE' named entity 'CARDIAC MYOCYTES' named entity 'CELL GROWTH' named entity 'CENTRAL' named entity 'RECEPTOR' named entity 'ANGIOTENSIN II' named entity 'CELLULAR' named entity 'FIBROBLASTS' named entity 'HYPERTENSION' named entity 'angiotensin-converting enzyme 2' named entity 'bioactive' named entity 'summarizes' named entity 'vascular' named entity 'Peptides' named entity 'Angiotensin II' named entity 'central product' named entity 'receptor' named entity 'angiotensin II' named entity 'Ang II' named entity 'renin-angiotensin' named entity 'cardiovascular diseases' named entity 'hypertension' named entity 'smooth muscle cells' named entity 'Angiotensin II' named entity 'thickening' named entity 'central nervous' named entity 'amino acid residues' named entity 'acute myocardial infarction' named entity 'pressor' named entity 'renin' named entity 'sympathetic neurons'

Faceted Search & Find service v1.13.91 as of Mar 24 2020

Alternative Linked Data Documents: Sponger | ODE Content Formats:

RDF

ODATA

Microdata

About

OpenLink Virtuoso version 07.20.3229 as of Jul 10 2020, on Linux (x86_64-pc-linux-gnu), Single-Server Edition (94 GB total memory)
Data on this page belongs to its respective rights holders.
Virtuoso Faceted Browser Copyright © 2009-2024 OpenLink Software