About: Viral pathophysiology of multiple sclerosis: A role for Epstein-Barr virus infection?

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An Entity of Type : schema:ScholarlyArticle, within Data Space : covidontheweb.inria.fr associated with source document(s)

Attributes	Values
type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
has title	Viral pathophysiology of multiple sclerosis: A role for Epstein-Barr virus infection?
Creator	Bosca, Isabel Dobson, Ruth Etemadifar, Masoud Giovannoni, Gavin Maggiore, Cosimo Maghzi, Amir-Hadi Marta, Monica Meier, Ute-Christiane
Source	Elsevier; Medline; PMC
abstract	Abstract Multiple sclerosis (MS) is the most common inflammatory demyelinating and degenerative disease of the CNS. The cause of MS is unknown but environmental risk factors are implicated in MS. Several viruses have been proposed as a trigger for MS, and lately Epstein-Barr virus (EBV) has become the leading candidate. An infectious aetiology fits with a number of epidemiological observations in addition to the immunopathological features of the disease. In this review we will summarize the emerging evidence, which demonstrates a strong association between EBV infection and MS. The conundrum remains as to whether EBV is directly involved in the pathophysiology of MS, or alternatively if the immunopathology of MS somehow affects the regulation of EBV infection.
has issue date	2011-02-28(xsd:dateTime)
bibo:doi	10.1016/j.pathophys.2010.04.003
bibo:pmid	20538440
has license	els-covid
sha1sum (hex)	0d5c950f77a1ac543d72adf1323703d0d0134c9f
schema:url	https://doi.org/10.1016/j.pathophys.2010.04.003
resource representing a document's title	Viral pathophysiology of multiple sclerosis: A role for Epstein-Barr virus infection?
has PubMed Central identifier	PMC7135674
has PubMed identifier	20538440
schema:publication	Pathophysiology
resource representing a document's body	covid:0d5c950f77a1ac543d72adf1323703d0d0134c9f#body_text
is schema:about of	named entity 'EBV' named entity 'conundrum' named entity 'summarize' named entity 'INFECTIOUS' named entity 'UNKNOWN' named entity 'MULTIPLE SCLEROSIS' named entity 'VIRUSES' named entity 'STRONG' named entity 'PATHOPHYSIOLOGY' named entity 'NUMBER OF' named entity 'HAVE' named entity 'INFLAMMATORY' named entity 'CAUSE' named entity 'EPSTEIN-BARR VIRUS INFECTION' named entity 'VIRAL' named entity 'MULTIPLE SCLEROSIS' named entity 'PATHOPHYSIOLOGY' named entity 'ROLE' named entity 'EVIDENCE' named entity 'ADDITION' named entity 'CNS' named entity 'BUT' named entity 'EPIDEMIOLOGICAL' named entity 'FITS' named entity 'EBV INFECTION' named entity 'DISEASE' named entity 'EBV' named entity 'MS.' named entity 'COMMON' named entity 'FEATURES' named entity 'ASSOCIATION' named entity 'CANDIDATE' named entity 'REGULATION' named entity 'REVIEW' named entity 'AETIOLOGY' named entity 'ENVIRONMENTAL RISK FACTORS' named entity 'TRIGGER' named entity 'PROPOSED' named entity 'IMMUNOPATHOLOGY' named entity 'LEADING' named entity 'DIRECTLY' named entity 'EPSTEIN-BARR VIRUS' named entity 'INVOLVED' named entity 'OBSERVATIONS' named entity 'DEGENERATIVE DISEASE' named entity 'observations' named entity 'epidemiological' named entity 'aetiology' named entity 'viruses' named entity 'EBV infection' named entity 'Epstein-Barr virus infection' named entity 'remains' named entity 'hybridisation' named entity 'genome' named entity 'EBV infection' named entity 'lytic' named entity 'pediatric' named entity 'EBV' named entity 'clinical remission' named entity 'high levels' named entity 'NAb' named entity 'HLA' named entity 'CIS' named entity 'viruses' named entity 'EBV' named entity 'MHC class I molecules' named entity 'antigen' named entity 'IgG' named entity 'antibody' named entity 'saliva' named entity 'memory B cells' named entity 'Cytotoxic' named entity 'peripheral blood mononuclear cells' named entity 'EDSS'

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