About: Inflammasome in the Pathogenesis of Pulmonary Diseases

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An Entity of Type : schema:ScholarlyArticle, within Data Space : covidontheweb.inria.fr associated with source document(s)

Attributes	Values
type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
has title	Inflammasome in the Pathogenesis of Pulmonary Diseases
Creator	Fan, Jie Shi, Xueying Wen, Zongmei Xu, Fengying
Source	PMC
abstract	Lung diseases are common and significant causes of illness and death around the world. Inflammasomes have emerged as an important regulator of lung diseases. The important role of IL-1 beta and IL-18 in the inflammatory response of many lung diseases has been elucidated. The cleavage to turn IL-1 beta and IL-18 from their precursors into the active forms is tightly regulated by inflammasomes. In this chapter, we structurally review current evidence of inflammasome-related components in the pathogenesis of acute and chronic lung diseases, focusing on the “inflammasome-caspase-1-IL-1 beta/IL-18” axis.
has issue date	2018-09-05(xsd:dateTime)
bibo:doi	10.1007/978-3-319-89390-7_6
bibo:pmid	30536170
has license	no-cc
sha1sum (hex)	18e5ca6859a1131329eb3b2c61b66955d97aed20
schema:url	https://doi.org/10.1007/978-3-319-89390-7_6
resource representing a document's title	Inflammasome in the Pathogenesis of Pulmonary Diseases
has PubMed Central identifier	PMC7123416
has PubMed identifier	30536170
schema:publication	Inflammasomes: Clinical and Therapeutic Implications
resource representing a document's body	covid:18e5ca6859a1131329eb3b2c61b66955d97aed20#body_text
is schema:about of	named entity 'Pathogenesis' named entity 'fibrogenic' named entity 'IL-18' named entity 'effector' named entity 'ATP' named entity 'caspase-1' named entity 'IL-1Ra' named entity 'fibrosis' named entity 'cryptogenic fibrosing alveolitis' named entity 'inflammasome' named entity 'lung injury' named entity 'murine' named entity 'NLRP3' named entity 'IL-1' named entity 'MyD88' named entity 'wild-type' named entity 'mice' named entity 'influenza virus' named entity 'antibiotics' named entity 'mitochondrial' named entity 'bronchial epithelial cells' named entity 'pneumonia' named entity 'high uric acid levels' named entity 'epithelial cells' named entity 'mycobacterial' named entity 'bleomycin' named entity 'Nlrp3' named entity 'infection' named entity 'pulmonary diseases' named entity 'caspase activation' named entity 'caspase-1' named entity 'epithelial cells' named entity 'NLRC4' named entity 'mice' named entity 'neutrophil' named entity 'transcription factor' named entity 'bleomycin' named entity 'inflammasome' named entity 'gene' named entity 'NLRP3' named entity 'chronic inflammatory' named entity 'IL-18' named entity 'caspase-1' named entity 'Lung diseases' named entity 'IL-1' named entity 'NLRP3' named entity 'Pseudomonas aeruginosa' named entity 'antigen' named entity 'Mycobacterium kansasii' named entity 'antigenic' named entity 'streptolysin O' named entity 'lung infection' named entity 'Il1r1' named entity 'adaptive immunity' named entity 'Serum' named entity 'NLRC4' named entity 'ovalbumin' named entity 'COPD' named entity 'NLRP3' named entity 'immunoglobulin' named entity 'intracellular' named entity 'IL-1' named entity 'Klebsiella pneumoniae' named entity 'secretion' named entity 'fibrosis' named entity 'lungs' named entity 'dectin-1' named entity 'motility' named entity 'NLRP3' named entity 'gene knockout' named entity 'lung parenchyma' named entity 'inflammasomes' named entity 'DAMPs' named entity 'lipopolysaccharides' named entity 'secretion' named entity 'mice' named entity 'lung injury' named entity 'silica'

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